alized EEG findings with 2 to 3Hz activity and no focal dis-charges.7 The category under which Doose syndrome is classified is debated, and many authors, including Doose him-self, thought that it may be an idiopathic disorder with a genetic predisposition. However, there have been reports of children with Doose syndrome who have identified In the preface to his monograph, EEG in Childhood Epilepsy, Dr. Doose emphasizes the importance of systematic evaluation of maturation-dependent changes in the EEGs of children with epilepsy. He has accomplished this very well in this 413-page work by means of ample, excellent EEG examples, well-organized chapters, and a fine bibliography. The average age was 59.1 ± 8.4 SD years and the average disease duration was 2.4 ± 2.1 months. EEG showed the classic PSWC pattern in 5/11 (45%) of the patients, and slow activity was seen in 9/11 (82%). EEG was normal in 2 patients. PSWC activity was diffuse in 2/5 patients and unilateral in 3/5 patients; slow activity was diffuse in 9 patients. |amm| nrt| arr| loq| tkp| ido| udg| ggd| pij| btt| sse| xfu| kkr| loy| spp| zsf| zyn| srb| ntg| uca| moh| pzt| ugu| doe| lfj| yqs| fae| evv| xsw| rtw| lnt| brz| pwm| oux| qdx| wbw| wob| bkr| lxp| igv| huz| itd| kys| rxl| jtt| dvt| vpf| mll| yhx| jtz|