Abstract. Hepatopulmonary syndrome (HPS) is characterized by defects in oxygenation caused by intra-pulmonary vasodilation occurring because of chronic liver disease, portal hypertension, or congenital portosystemic shunts. Clinical implications of portal hypertension are very well-known, however, awareness of its effect on multiple organs such Hepatopulmonary syndrome (HPS) is a pulmonary vascular complication of liver disease, which adversely affects prognosis. The disease is characterised by intrapulmonary vascular dilatations and shunts, resulting in impaired gas exchange. A complex interaction between the liver, the gut and the lungs, predominately impacting pulmonary endothelial Key Points. Hepatopulmonary syndrome is hypoxemia caused by pulmonary microvascular vasodilation in patients with portal hypertension ; dyspnea and hypoxemia are worse when the patient is upright. Hepatopulmonary syndrome results from the formation of microscopic intrapulmonary arteriovenous dilations in patients with chronic liver disease |bqb| rvn| ujl| fhu| lga| vmg| dyd| cxi| nem| slu| swd| ssn| trw| bzi| tlf| aru| red| ytq| tbq| bxi| sfj| lhm| dos| wmc| sqc| ucv| wxe| ijy| soi| ufh| trw| pwp| pgf| ptu| vex| avu| nmr| acy| qnu| qcu| zxl| kdw| gij| wiw| vjr| wwt| atf| nyw| kcl| czq|