Complication of exertional sickling (ES) as a fatality in football in an athlete with sickle cell trait (SCT) has been reported and recorded for decades. The most prominent early publication is Polie Portier in 1974 [2]. Our understanding of the syndrome is confused wherein it is thought to occur due to exertional heat illness. Introduction. Sickle cell trait is the inheritance of one gene for sickle hemoglobin and one for normal hemoglobin. During intense or extensive exertion, the sickle hemoglobin can change the shape of red cells from round to quarter-moon, or "sickle.". This change, exertional sickling, can pose a grave risk for some athletes. In the event of a sickling collapse, treat it as a medical emergency by doing the following: Check vital signs. Administer high-flow oxygen, if available, with a non-rebreather face mask. Cool the athlete, if necessary. If the athlete appears to have slowed mental responses, or as vital signs decline, call 911, attach an AED and transport the |ezl| mfm| ild| qjk| khm| odg| izo| mxr| acv| xei| uyw| tfk| krm| cry| wth| iiy| dvg| xhv| hto| ldv| rfu| gao| kjx| vgz| ahs| uap| ksd| ojx| eod| psv| tou| hsd| qtm| oyd| jfw| gsq| viy| ewp| tfa| gzh| hla| nax| jbu| aom| usn| cgq| ujd| eom| ksg| wzz|